Treatment of Craniomaxillofacial Fibrous Dysplasia
نویسندگان
چکیده
منابع مشابه
Treatment of Craniomaxillofacial Fibrous Dysplasia
Fibrous dysplasia is a benign osseous disease, although radical resection is commonly recommended because of the possibility of recurrence or malignant change. However, radical resection is undesirable, particularly in the maxillofacial region, because facial bones are difficult to replace in an aesthetically-pleasing manner after radical excision. In our department, conservative shaving, as re...
متن کاملCraniomaxillofacial fibrous dysplasia: a 10-year database 1996-2006.
Fibrous dysplasia is a rare bone disease caused by an abnormal proliferation of fibrous tissue in bone. We retrospectively evaluated eight patients (female to male ratio 3:1, mean age 22.5 years, range 10-32) with a monostotic form who were treated between 1996 and 2006. Two each were affected in the lower jaw, the upper jaw, the midface, and the frontoparietal region. Most patients were referr...
متن کاملCraniomaxillofacial Fibrous Dysplasia: Conservative Treatment or Radical Surgery? Report of Two Cases
Introduction: Fibrous Dysplasia is a rare bone disease wherein the normal bone is replaced by fibrous tissue causing functional and aesthetic problems. They pose difficulty in diagnosis, treatment planning. Case Report: We are presenting 2 cases of Fibrous Dysplasia wherein the treatment done was resection of body of mandible with reconstruction with iliac graft in the first case and a shave do...
متن کاملCraniofacial Fibrous Dysplasia
Fibrous dysplasia (FD) is an osseous growth dis-order, producing immature bone and characte-rized by the replacement of normal bone with fibro-osseous connective tissue. It is a bone dys-plasia that has the potential to cause significant cosmetic and functional disturbances, particularly in the craniofacial skeleton. Cra-niofacial fibrous dysplasia is one of the three types of polyostotic fibro...
متن کاملDenosumab treatment for fibrous dysplasia.
Fibrous dysplasia (FD) is a skeletal disease caused by somatic activating mutations of the cyclic adenosine monophosphate (cAMP)-regulating protein, α-subunit of the Gs stimulatory protein (G(s) α). These mutations lead to replacement of normal bone by proliferative osteogenic precursors, resulting in deformity, fracture, and pain. Medical treatment has been ineffective in altering the disease ...
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ژورنال
عنوان ژورنال: Surgical Science
سال: 2015
ISSN: 2157-9407,2157-9415
DOI: 10.4236/ss.2015.66036